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Superoxide Dismutase, Catalase,
& Glutathione Protect Blood Cells
From Oxidative Stress & Damage
Several powerful oxidants are produced during the
course of metabolism, in both blood cells and most
other cells of the body. These include superoxide O2
hydrogen peroxide (H2O2), peroxyl radicals ROO
and hydroxyl radicals (OH•). The last is a particularly
reactive molecule and can react with proteins, nucleic
acids, lipids, and other molecules to alter their structure
and produce tissue damage. The reactions listed in
Table 52–4 play an important role in forming these oxidants
and in disposing of them; each of these reactions
will now be considered in turn.

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دانلود کتاب بیوشیمی هارپر به زبان اصلی

Harper’s
Illustrated
Biochemistry

a LANGE medical book
twenty-sixth edition
Robert K. Murray, MD, PhD
Professor (Emeritus) of Biochemistry
University of Toronto
Toronto, Ontario
Daryl K. Granner, MD
Joe C. Davis Professor of Biomedical Science
Director, Vanderbilt Diabetes Center
Professor of Molecular Physiology and Biophysics
and of Medicine
Vanderbilt University
Nashville, Tennessee
Peter A. Mayes, PhD, DSc
Emeritus Professor of Veterinary Biochemistry
Royal Veterinary College
University of London
London
Victor W. Rodwell, PhD
Professor of Biochemistry
Purdue University
West Lafayette, Indiana
Lange Medical Books/McGraw-Hill
Medical Publishing Division
New York Chicago San Francisco Lisbon London Madrid Mexico City
Milan New Delhi San Juan Seoul Singapore Sydney Toronto

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The Red Blood Cell Has a Glucose Transporter in Its Membrane The entry rate of glucose into red blood cells is far greater than would be calculated for simple diffusion. Rather, it is an example of facilitated diffusion (Chapter 41). The specific protein involved in this process is called the glucose transporter or glucose permease. Some of its properties are summarized in Table 52–3. The process of entry of glucose into red blood cells is of major importance because it is the major fuel supply for these cells. About seven different but related glucose transporters have been isolated from various tissues; unlike the red cell transporter, some of these are insulindependent (eg, in muscle and adipose tissue). There is considerable interest in the latter types of transporter because defects in their recruitment from intracellular sites to the surface of skeletal muscle cells may help explain the insulin resistance displayed by patients with type 2 diabetes mellitus.

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